Minor chorea in adults. Lesser chorea: causes, symptoms, treatment

Usually, when a child gets sick, the mother sees it immediately. But there are diseases that cannot be easily distinguished from simple self-indulgence or indiscipline. This exceptional disease is called "chorea" - for which children are more often punished than shown to a doctor.

Chorea: what is it?:

Chorea (minor chorea, Witt's dance, rheumatic or infectious chorea, Sydenham's chorea) is a neurological course of rheumatic disease. Accompanied by disease motor disorders, uncontrollable muscle contractions and psycho-emotional disorders.

It is now 100% known that the disease is caused by group A β-hemolytic streptococcus. This microorganism affects upper section respiratory system, causing tonsillitis with sore throat. The body begins to fight the infection by producing antibodies to streptococcus to fight it. Some people experience a cross-immune response, i.e. antibodies begin to attack their own body cells - the brain ganglia in the head, joints, heart muscle, kidneys, etc. A rheumatic infection in the organs and inflammation of the subcortical layer of the brain begins to develop, which manifests itself in specific symptoms.

Development streptococcal infection with brain damage does not manifest itself in everyone. The main predisposition factors are:

Heredity;
- violations hormonal levels;
- chronic diseases upper respiratory tract;
- carious teeth;
- disturbances in the functioning of the immune system;
- natural increased nervous excitability and emotionality;
- asthenic body type.

Chorea affects children of preschool and school age. Under the age of 3 and after 15 years, the disease practically does not occur. There is also a greater predisposition to the disease in girls than in boys.

The course of chorea and its symptoms:

Chorea develops gradually after tonsillitis, scarlet fever, tonsillitis or flu. All main signs can be classified into 4 groups:

1. hyperkinesis (involuntary and uncontrolled muscle movements);

2. lack of coordination (impaired coordination of movements);

3. hypotension (muscle weakness);

4. abrupt change moods.

First of all, the child is overwhelmed by absent-mindedness, tearfulness and resentment. Clarity and coordination are lost in movements. Handwriting deteriorates in school-age children, preschool age– drawings lose clarity. The child begins to eat sloppily, difficulties arise with holding objects, and grimaces are seen on the face. The more attentive parents or teachers are at school, the sooner they will be able to understand that the child is not playing around, and his entire condition is explained by a specific disease.

Rheumatic lesions internal organs after chorea can appear after a very long time long time, up to several years.

Impaired movement of the limbs manifests itself in erratic tremors due to muscle damage. The movements are completely involuntary, but intensify with additional stimuli and stop when the child falls asleep. The twitching progresses quite quickly, and at the peak of the disease it seems that the child is in constant motion. Legs, arms, shoulders - everything is covered in meaningless and unnecessary contractions. The child cannot walk or stand normally, and his speech is impaired. Conscious movements are fleeting and practically imperceptible (squeezing a hand, holding objects, etc.). In parallel with twitching, muscle hypotonia is noted, i.e. when lifting the patient lying down, holding the armpits, the shoulders involuntarily fall back, becoming limp.

With the development of chorea in mild form, the main symptom is not a movement disorder, but hypotension, which is immediately perceived as paresis. There is also an uncontrollable change in mood for no reason: the child becomes easily vulnerable, laughter quickly gives way to crying or irritability.

If the diaphragm is affected during the disease, Czerny's symptom or “paradoxical breathing” is noted. This is manifested by retraction of the abdominal wall during inspiration, instead of normal protrusion.

As a rule, the disease proceeds without increase general temperature bodies. Fever is possible with exacerbation of rheumatic inflammation of internal organs.

The disease lasts about 7-10 weeks, but can drag on for up to 4 months. Relapses are common, as well as regular changes from remission to worsening health conditions.

The prognosis is usually favorable and the patient usually recovers. There is some dependence on the speed of development of clinical signs: the slower the symptoms of the disease appear and the deeper the muscle damage, the longer the recovery will take. Relapses are observed after tonsillitis and rheumatic exacerbations.

How to diagnose?:

As already noted, it is difficult to immediately determine the disease based on clinical signs alone, especially at the beginning of its appearance. As the disease progresses experienced pediatrician makes a diagnosis quickly and accurately.

People with this disease are referred to pediatric neurologist(or the pediatrician sends him to him). The doctor carefully studies the medical history, examines the child and performs a number of diagnostic procedures (blood tests and neurological tests). A blood test determines the presence of streptococcal infection and potential rheumatic damage to the body.

Can be assigned:

Electroencephalogram, computer or magnetic resonance imaging to analyze the functioning of the brain;

Cerebrospinal fluid analysis;

Electromyography to determine dysfunction skeletal muscles.

Chorea must be differentiated from dysmetabolic encephalopathies, classic tics and viral encephalitis.

When making a diagnosis, the doctor must perform the following neurological tests:

-“Filatov’s eyes and tongue” or “chameleon tongue”(the patient cannot hold his tongue out eyes closed mi);

- Gordon phenomenon(during the knee reflex test, the lower leg lowers after rising only a few seconds later, freezing in the air and making several swings before stopping);

- pronator sign(when you raise your palms above your head, forming a semicircle with your hands and a candle with your hands, an involuntary turning of the palms outward is noted);

- "trochaic brush"(extended hands are bent at the radial and wrist joints with the fingers extended and pressed to the palm thumb);

- flabby shoulder syndrome(when lifting the patient's armpits, there is some sinking of the head into the shoulders).

Treatment of chorea in children:

Acute course chorea is cured an order of magnitude faster than sluggish disease, which can last up to 12 months.

The child needs peace and long sleep, for which purpose “sleeping wards” are organized in the hospital with a ticking clock or, for example, a ticking chronometer, as well as with windows open to the sun. This is done due to the fact that during sleep hyperkinesis is completely eliminated, and the child can be completely at rest at this time.

Shown their effectiveness physiotherapy, physiotherapy, as well as creative activities where you need to work with your fingers (embroidery, modeling, knitting, drawing, cutting, etc.).

As drug therapy are appointed:

Antirheumatic drugs;

Antibiotics;

Drugs that inhibit nervous excitability(neuroleptics, antidepressants and sleeping pills);

Hormonal agents;

B vitamins.

In case of insufficient efficiency medicines A psychologist helps to combat psycho-emotional changes.

Conclusions:

Minor chorea in children does not directly threaten the child’s life (frequency deaths from rheumatic complications is up to 1%), and with quality treatment may go into long-term remission or be cured completely. Walkthrough full course antibiotic therapy against streptococci - the main preventative measure from illness. It is also important to remember a few important rules:

1. Chorea often appears after scarlet fever, flu and sore throat, so children after illness should be under their mother’s close supervision.

2. You should immediately seek medical advice if you notice mood swings, muscle twitching or uncoordinated movements.

3. When making a diagnosis, doctors' instructions must be strictly followed.

Chorea is pathological condition, characterized by the development of hyperkinesis various muscles bodies. A person makes aimless, sweeping, chaotic and jerky movements, mainly with the upper and lower limbs. The disease is caused by damage to the basal ganglia of the brain, as well as the dentate nuclei of the cerebellum. This happens under the influence of various factors.

In most clinical situations, the process also involves reticular formation brain Because of all this pathological changes violated physiological process transmission of dopamine (neurotransmitter). As a result, this leads to wide and erratic muscle movements. Treatment of the pathology will be long and complex. The sooner the first symptoms are noticed and the correct treatment, the more favorable the prognosis will be.

Forms

• hereditary forms. This includes. This hereditary disease There are two types: chronic late start and non-progressive with early onset;
• chorea in extrapyramidal diseases. This type often develops with Lesch-Nyhan syndrome, hepatocerebral dystrophy;
• secondary forms. They occur when the basal structures of the brain are damaged. The reasons may be different - infection, trauma, etc. Secondary forms include minor chorea. It develops due to streptococcal infection entering the circulatory system. The source can be carious teeth and inflamed tonsils. “Target organs” for infection: joints, heart valves, brain. The disease is in the majority clinical cases affects young children whose bodies are weakened. It is characterized by a relapsing course.

Causes

• malfunction of the immune system;
• burdened heredity. This is the cause of the development of Huntington's chorea ( genetic disease). The first symptoms of pathology in the body may appear only at the age of 40. Before this, a person will not even be able to suspect that he is sick. Hyperkinesis appears against the background of emotional instability and developing dementia;
• brain injuries;
• cerebral palsy;
• infectious diseases that have bacterial or viral nature, such as encephalitis, meningitis, etc.;
• diseases of the blood vessels supplying the brain with blood;
• intoxication of the body;
• metabolic disorders (bilirubin encephalopathy);
• (in this case the person develops rheumatic chorea);

The most common types of disease are:

• It can appear in a person at any age, but, as a rule, it most often affects the body at 35–40 years of age. It is characterized by choreic hyperkinesis, dementia and personality disorders. The disease develops gradually and in its first stages it is almost impossible to notice hyperkinesis. Chaotic twitching can be noted in the facial area. The person frowns or opens his mouth, licks his lips, sticks out his tongue. Next symptom- “fingers playing the piano.” Afterwards, gait and static behavior change, speech and swallowing are impaired. Memory deteriorates and dementia develops. A person stops basicly serving himself. It is important to notice the first symptoms of the disease in a timely manner in order to consult a specialist and carry out adequate treatment;
• Sydenham's chorea or rheumatic chorea. The disease develops some time after a streptococcal infection or an exacerbation of rheumatism. The disease in most clinical cases is observed in childhood and adolescence. Most often it is girls who get sick. The symptoms of the disease are clearly expressed. At the first stages of development of the pathology, motor disinhibition with grimacing and exaggerated gestures is noted. If generalized hyperkinesis is more pronounced, then the child completely loses the ability to care for himself, dysarthria develops and respiratory function is impaired. All this makes it impossible for him to communicate and move. In some cases, in the generalization stage of hyperkinesis, hemichorea occurs. The appearance of muscle hypotonia is characteristic. Hyperkinesis regresses over three to six months. Chorea in children is more severe than in adults;
• Chorea of ​​pregnancy. The disease develops in primiparous women who suffered rheumatic chorea in childhood. The pathology is associated with antiphosphorlipid syndrome. The disease usually develops at 3–5 months of pregnancy, and can also recur with next pregnancies. Symptoms occur spontaneously over several months.

Symptoms

In medicine, there are several symptoms that are characteristic of any type of chorea:

• involuntary movements of the limbs (an important diagnostic symptom);
• head twitching;
• decreased muscle tone;
• impaired coordination of movements;
• grimacing;
• gesticulation is increased;
• if you ask a person to write something, you can notice a change in handwriting;
• memory loss;
• hyperkinesis in this disease disappears during sleep;
• dancing gait;
• temperature increase;
• all the movements that a person tries to control, he performs with great difficulty.

Diagnostics

Diagnosis of chorea is based on the clinical picture of the disease and patient complaints. For getting additional information use laboratory and instrumental techniques:

• . This method allows you to detect a decrease or increase in blood levels. This is typical for this disease. The blood is also tested to detect streptococcal infections in the patient's body. For this purpose, pay attention to indicators C-reactive protein, rheumatoid factor (especially important indicator for the diagnosis of rheumatic chorea);
• electroencephalogram. The technique allows you to detect even minor changes in brain activity;
• electromyography. An innovative technique that allows you to thoroughly study the biopotentials of muscles. With this disease they will be lengthened;
• CT scan.

When examining a patient, the doctor pays attention to the most characteristic symptoms of this disease - hyperkinesis and grimacing. The presence of these two signs already indicates that the person has experienced a malfunction nervous system. The treatment regimen will be selected based on the data obtained during the examination.

Treatment

Treatment of chorea should only be carried out by a highly qualified doctor who can correctly select necessary plan therapeutic measures. He will also select the appropriate medications and their dosage. Throughout the treatment period, therapy may vary slightly, depending on the improvement or deterioration of the patient's condition.

Drug therapy plays a role in treating the disease key role. The patient is prescribed the following groups of drugs:

• neuroleptics. Most doctors prefer haloperidol. It is often combined with reserpine and aminazine. The treatment is highly effective with a correctly selected regimen;
• antibiotics. This group synthetic medical supplies used if the disease was caused by an infection. The drugs of choice are bicillin, penicillin;
• sedatives. Preference is given to the group of barbiturates;
• corticosteroids;
• NPPP;
• antihistamines;
• vasodilators.

In combination with drug therapy use vitamin therapy, as well as physiotherapeutic treatment, which can reduce unpleasant symptoms. Good effect have radon baths, galvanization, electrosleep.

Is everything correct in the article? medical point vision?

Answer only if you have proven medical knowledge

Diseases with similar symptoms:

Minor chorea (Sydenham's chorea) is a disease of rheumatic etiology, the development of which is based on damage to the subcortical nodes of the brain. A characteristic symptom of the development of pathology in humans is a violation motor activity. The disease mainly affects young children. Girls get sick more often than boys. Average duration illness – from three to six months.

Chorea minor (Sydenham's chorea) is the main form of rheumatic damage to the nervous system in children. Often the first clinical sign rheumatism, but can develop against the background of a latent current disease or during its inter-relapse period. Children aged 5 to 15 years are most often affected (girls are approximately 2 times more likely than boys).

The etiology and pathogenesis of X. m. are inseparable from others rheumatic lesions. The main etiological role is played by streptococcal infection, which is considered as a trigger mechanism of the disease, including a number of immunological ones, in particular allergic mechanisms. In the formation of pathological reactivity, a significant role belongs to disorders of the adaptation system hypothalamus - pituitary gland - adrenal glands. Humoral and cellular immunological disorders are manifested by an increase in the titer of antistreptolysin-O, antistreptohyaluronidase and antistreptokinase.

Pathoanatomical changes are degenerative-toxic and inflammatory nature. The main changes are localized in the subcortical nuclei and superior cerebellar peduncles; The small cells of the lentiform nucleus are most affected. The cortex, red nuclei, substantia nigra, cerebellum and other structures are also affected. Vasculitis with fibrinoid swelling and hyalinosis are noted vascular wall, degenerative changes neurons, microglial reaction.

Clinical picture. Hm. usually develops gradually against the background normal temperature bodies. Febrility in some patients occurs due to other rheumatic lesions. TO initial manifestations First of all, it refers to cerebrasthenic syndrome. Patients experience irritability, mild excitability, and emotional instability. Motor disinhibition and awkwardness of movements gradually increase, objects begin to fall out of hands, and handwriting in school-age children deteriorates significantly. After about 1-2 weeks, hyperkinesis of facial muscles and limbs appears. The patient wrinkles his forehead, closes his eyes, and stretches out his lips. Choreic hyperkinesis in the limbs is fast, impetuous, wide-ranging, and non-stereotypical. IN initial period disease, it is more pronounced in proximal parts arms and legs. At rest, hyperkinesis decreases or disappears, and with excitement, physical stress, fatigue increases significantly. In the most severe cases a “motor storm” occurs - the patient is in motion all the time: he is thrown up, sometimes he cannot stay in bed, hits himself, receives multiple bruises, is deprived of the ability to perform purposeful movements, cannot bring food to his mouth, walk, stand, sit . Speech becomes dysarthric, some syllables or words are pronounced too loudly, others in a whisper. However, in many cases, hyperkinesis with X. m. is not clearly expressed and they have to be identified special techniques. In this case, the simplest and most convenient test is Yuretskaya and Shanko: a child in the Romberg position is asked to stretch out his arms, spread his fingers, close his eyes and stick out his tongue. This test can also be carried out with a dosed physical activity(squatting, running in place, jumping on one leg). As a result of such an examination, it is possible to detect general motor restlessness, twitching of the tongue and fingers.

A constant sign of the disease is muscle hypotonia, up to atony with mild chorea. In these cases, active movements and the implementation of hyperkinesis become impossible. Arises clinical picture pseudoparalysis. IN in some cases the child cannot even hold his head up. The appearance of hyperkinesis in patients with X. m. indicates the positive dynamics of the disease. The cause of the development of muscle hypotonia is functional-dynamic disorders of the descending connections of the limbic-reticular system.

Along with hyperkinesis and muscle hypotonia Psychotic disorders with delusions, hallucinations, and motor agitation may occur. These patients require hospitalization in a psychiatric department.

At H.m. others are noted neurological symptoms, for example, the “eyes and tongue” symptom, when the patient cannot simultaneously keep his eyes closed and his tongue out; Czerny's symptom - a violation of the synergistic movements of the diaphragm and intercostal muscles during breathing, and therefore sinks when exhaling abdominal wall. Changes are happening tendon reflexes(with mild chorea they do not disappear); knee reflexes are pendulum-like in nature or when they are evoked, the leg freezes in the extension phase in knee joint. Sometimes hypertension syndrome is observed, accompanied by headache, vomiting, and slight changes in the fundus. In most patients, H.m. are determined functional changes cardiac activity, which then quickly disappear. Some patients have myocarditis or endocarditis, and therefore patients require cardiac evaluation. With a recurrent course of X. m., changes in the heart occur more often and are more severely expressed than at the onset of the disease. The high vascular permeability characteristic of rheumatism is also expressed in chorea. It is detected by checking the symptoms of a pinch or tourniquet, as well as by a cup test. Changes biochemical tests And general analysis blood levels during chorea are determined by the course of the main rheumatic process.

At correct mode and treatment of X. m. lasts about 2 months, mild chorea lasts up to 5-6 months. With more acute development and pronounced hyperkinesis regression symptoms are coming faster than in subacute development and mildly expressed neurological disorders. In approximately half of patients, cancer occurs with relapses, which often occur after 1-2 years; in some patients, multiple relapses are observed after a short period of time (continuously relapsing course). Typically, relapse is preceded by a sore throat or exacerbation of the rheumatic process. In all cases, relapse should be considered as a manifestation of the activity of rheumatism.

Treatment. The main principles of therapy are the combination of antirheumatic drugs with sedatives. Installed bed rest, psycho-traumatic factors are eliminated, it is ensured good nutrition. Antirheumatic drugs include salicylates, butadione, and brufen in age-related doses. Average duration treatment with these drugs for 4 weeks. Penicillin and ampicillin are also prescribed in normal doses. Antihistamines are indicated (suprastin, tavegil, fenkarol, etc.), and it is advisable to change medications every 7-10 days. If therapy is ineffective and in case of relapse, glucocorticoids (prednisone, dexazone, dexamethasone) are indicated; maximum dose These drugs are given for 7-10 days. The total duration of treatment is 40 days. Large doses indicated ascorbic acid(0.1-0.2 g 2-3 times a day), B vitamins. Prescribed sedatives: bromides, valerian preparations, phenobarbital, phenibut. For severe hyperkinesis, haloperidol and chlorpromazine are prescribed for several days. For any form of rheumatism, sanitation of a possible focus of streptococcal infection (tonsils, carious teeth, sinusitis) is indicated. Surgery in these cases, it is carried out in the interictal period.

Chorea minor is a rheumatic abnormality. It is expressed in bright, uncontrollable movement disorders. The nature of this nervous pathology I have baffled doctors more than once. Today, doctors have come to the conclusion that the anomaly develops against a rheumatic background.

The main root cause of the development of this anomaly is a relapse of scarlet fever or tonsillitis.

Factors in the development of the disease

It is noteworthy that girls are at greater risk of developing chorea minor than boys. The reasons for the development of this disease have long been a real mystery to doctors. Now doctors have come to the conclusion that the root cause of the development of chorea minor is the penetration of streptococcal bacteria from group A into the body. The disease can begin to progress even against the background of tonsillitis, when bacteria, traveling through the blood, penetrate the connective tissue.

If infectious agent penetrates the central nervous system, then natural cause This is a disruption of the functioning of the GM. This leads to impaired coordination of movements and muscle tone.

The duration of the anomaly is 3−6 weeks.

Death is quite rare. The reason for this is damage to the cardiovascular system.

How does the pathology manifest itself?

The symptoms of chorea minor are quite specific and the disease can be diagnosed already at the dawn of its development.

Lesser chorea, being a “juvenile” anomaly, develops extremely rarely after puberty. Girls are again at risk. Minor chorea almost never occurs in adult young ladies. The exception is individuals who are prone to relapses during pregnancy.

Main signs

The main symptoms of minor chorea include uncontrolled motor passages. They are expressed in nervous twitching upper and lower limbs, which is called in medicine choreic hyperkinesis.

According to some researchers of this pathology, rheumatic cardiac anomalies can develop against the background of minor chorea.

The fact that girls are more susceptible to this disease is explained by the influence of female hormones.

Clinical signs

Subsequently, the symptoms of minor chorea manifest themselves in:

1. Muscle weakness.
2. Impaired motor coordination.
3. The presence of choreic hyperkinesis.

A sick child moves quickly, but not rhythmically. Motor passages are characterized by roughness, awkwardness and random distribution. From the outside it seems that the movements are very “ragged”.

Hyperkinesis can be either symmetrical or unilateral. They are expressed in facial expressions, feet, and hands. Laryngeal hyperkinesis is very common. Against this background, a person suffers slurred speech, swallowing is impaired.

The muscles of the trunk are rarely involved in hyperkinesis. In this case, rapid, almost interrupted breathing is observed. The severity of abnormal manifestations may vary. In some cases, there is a slight grimacing. Sometimes the patient suffers from violent pathological motor passages.

Diagnosis of the disease

You can pre-diagnose minor chorea immediately. To clarify the diagnosis, the specialist must collect an anamnesis of the patient’s life. For this purpose they are used special methods research. In addition, the blood of a person suffering from this disease is taken for laboratory testing. It is in the blood test that the number of markers of rheumatoid factor, as well as reactive protein and streptococcal infection is revealed.

The key research method is the electroencephalogram. This procedure assumes that GM activity is examined using waves. Then, to study skeletal muscles, the doctor prescribes electromyography to the patient.

For detection focal changes GM is prescribed CT.

Features of helping the patient

Treatment of minor chorea should be timely and correct. It is assumed that a person at risk, even before the occurrence of specific disorders is registered with the attending physician.

All instructions and wishes of the specialist must be strictly followed. In some cases, the doctor recommends surgery to remove the tonsils. It’s better to do this, because otherwise the child will get a sore throat very often.

Impulsive uncontrolled actions are stopped by taking antipsychotics.

Important to consider

Minor chorea is characterized by paroxysmal manifestations. The average duration of the abnormal process is twelve weeks. In some cases, the pathological period lasts twelve months.

Against this background, a sick child often complains of:

• lethargy and apathy;
• hyperfatigue;
• sleep disturbances (most often drowsiness is observed);
• hallucinations.

The “strange” behavior of a child is not always pampering.

If a child suddenly begins to wrinkle his forehead, spill the contents of a spoon before reaching his mouth, knock over the plate on himself and grimace, there is no need to rush to punish him or take him to a psychologist or priest. It is these signs that are the first signs in the development of chorea minor.

Therefore, if a child suffers from private sore throats, it is recommended to immediately consult a good pediatric doctor.

Conclusion

The prognosis for minor chorea, unfortunately, is not very encouraging. Patients often die from dementia or cachexia.

Prevention of the disease is possible. To do this, you need to promptly treat colds, and even better, prevent their occurrence.

Chorea minor is a disorder neurological origin, manifested by serious violations motor functions and psyche. Most often girls aged 6-15 years are affected. The disease strikes subcortical structures brain, especially the striatum. The course of the disease can be latent, subacute, acute and recurrent.

Symptoms

• Twitching of striated muscles of the face and body.
• Impaired coordination of movements.
• Mental changes.
• Paralysis.

The most important symptoms of minor chorea are sudden involuntary irregular movements of the limbs, head, torso and decreased muscle tone. With excitement, reflex movements intensify, but disappear during sleep. Mental changes may also appear. People around quickly notice that the child is nervous and often cries. It’s bad that adults incorrectly attribute involuntary twitching and reflex movements of facial muscles to mischief, because a disease that is not noticed in a timely manner can progress. The muscles of the limbs and face twitch faster, tremble, and cramp. Soon the child stops walking, sitting or speaking normally. Due to intense muscle contraction, dangerous exhaustion of the body can occur. In addition, the child’s mood worsens, he becomes quieter - these symptoms show that the patient is suffering due to his illness.

This pathology is very dangerous, because due to uncontrolled muscle contractions the patient can get injured, as a last resort even die from exhaustion.

Causes

The disease affects children with the so-called neuropathic constitution. It is assumed that minor chorea is a consequence of active rheumatism. This form of rheumatism appears only after a person has suffered infectious disease caused by a certain type streptococci. Rheumatic inflammation covers blood vessels, cortex and other brain structures and characteristic symptoms of chorea minor appear.

Adults can also get chorea. In addition to movement disorders, the main symptom of the disease is a progressive mental disorder. The disease begins at the age of 25-45, and people have to spend the rest of their lives in a psychiatric clinic or in a hospital with proper care. This is a hereditary form of the disease, although very rare.

Treatment of chorea minor

At the first symptoms of illness, consult a doctor immediately. This is very important if they appeared after active phase rheumatism.

After anamnesis, the doctor makes a diagnosis. True, at the beginning of the disease it is quite difficult to diagnose it. After production final diagnosis The doctor sends the child to the hospital.

Children diagnosed with chorea minor should be treated in hospital ( home treatment ineffective and only leads to loss of precious time). The disease can be cured with anti-inflammatory drugs, antibiotics, salicylates (hormones are often used). The patient especially needs intensive sedatives care

For children who have the above-mentioned symptoms, first of all, you need to try to understand and sympathize with them because of the difficulties that have befallen them. Do not mock under any circumstances.

Course of the disease

The disease can progress in different ways. In mild forms, the onset of pathology does not progress further, although such cases are rare. More often all the symptoms characteristic of this disease appear; sometimes it affects only one half of the body. This form of the disease is called hemichorea. The average duration of the disease is three months. If the child is treated and observed by a specialist, the prognosis is favorable. Improvement in the child’s condition can be determined by increasingly coordinated movements. True, sometimes the disease recurs, but its relapse can also be successfully treated. During a relapse, it is most often not the brain that is affected, but the heart muscle and valves, hence the heart defect.